    In the United States Court of Federal Claims
                                 OFFICE OF SPECIAL MASTERS
                                          No. 14-1025V
                                      Filed: August 18, 2017
                                        Not for Publication


*************************************
CHRISTOPHER PURVIS,                 *
                                    *
            Petitioner,             *
                                    *
 v.                                 *
                                    *
SECRETARY OF HEALTH                 *
AND HUMAN SERVICES,                 *
                                    *
            Respondent.             *
                                    *
*************************************


    ORDER GRANTING PETITIONER’S MOTION FOR RECONSIDERATION AND
                          CLARIFICATION 1

         On July 25, 2017, the undersigned issued a decision dismissing petitioner’s petition for
failure to make a prima facie case. On August 14, 2017, petitioner filed a Motion for
Reconsideration and Clarification. In his motion, petitioner asks the undersigned to reconsider
her July 25, 2017 decision, arguing that the undersigned did not properly consider the fact that
autoimmune medications were prescribed to petitioner. Petitioner further argues that petitioner’s
symptoms lasted more than six months and that there is no evidence that petitioner’s discitis
preceded his influenza vaccination. Petitioner also requests clarification of the undersigned’s
July 25, 2017 decision.



1
 Because this unpublished Order contains a reasoned explanation for the special master’s action in this
case, the special master intends to post this unpublished Order on the United States Court of Federal
Claims’ website, in accordance with the E-Government Act of 2002, 44 U.S.C. § 3501 note (2012)
(Federal Management and Promotion of Electronic Government Services). Vaccine Rule 18(b) states that
all decisions of the special masters will be made available to the public unless they contain trade secrets
or commercial or financial information that is privileged and confidential, or medical or similar
information whose disclosure would constitute a clearly unwarranted invasion of privacy. When such a
decision is filed, petitioner has 14 days to identify and move to redact such information prior to the
document’s disclosure. If the special master, upon review, agrees that the identified material fits within
the banned categories listed above, the special master shall redact such material from public access.
       Under Vaccine Rule 10(e)(2), the “special master may seek a response from the
nonmoving party, specifying both the method of and the timing for the response.” The
undersigned gave respondent the opportunity to respond to petitioner’s motion, but respondent’s
counsel said respondent did not plan on filing a response unless the undersigned ordered him to
and clarifying that his lack of response does not mean that respondent agrees with petitioner’s
motion.

       This matter is now ripe for adjudication.

       I.      Legal Standard

        A party seeking reconsideration must “support the motion by a showing of extraordinary
circumstances which justify relief.” Fru-Con Constr. Corp v. United States, 44 Fed. Cl. 298, 300
(Fed. Cl. 1999). A motion for reconsideration must be based upon a “manifest error of law, or
mistake of fact, and is not intended to give an unhappy litigant an additional chance to sway the
court.” Prati v. United States, 82 Fed. Cl. 373, 376 (Fed. Cl. 2008). Specifically, “the moving
party must show: (1) the occurrence of an intervening change in the controlling law; (2) the
availability of previously unavailable evidence; or (3) the necessity of allowing the motion to
prevent manifest injustice.” Matthews v. United States, 73 Fed. Cl. 524, 526 (Fed. Cl. 2006).
Where a party seeks reconsideration on the ground of manifest injustice, the party must be
mindful that “[m]anifest” means “clearly apparent or obvious.” Ammex, Inc. v. United States, 52
Fed. Cl. 555, 557 (Fed. Cl. 2002). Accordingly, a party cannot prevail on the ground of manifest
injustice unless the party demonstrates that the asserted injustice is “apparent to the point of
being almost indisputable.” Pac. Gas & Elec. Co. v. United States, 74 Fed. Cl. 779, 785 (Fed. Cl.
2006).

        A motion for reconsideration will not be granted if the movant “merely reasserts . . .
arguments previously made . . . all of which were carefully considered by the court.” Ammex,
52 Fed. Cl. at 557. Nor will a motion for reconsideration be granted if it is “based on evidence
that was readily available at the time” the matter was being decided. Seldovia Native Ass’n v.
United States, 36 Fed. Cl. 593, 594 (Fed. Cl. 1996). Finally, an evaluation of a motion for
reconsideration is to be “guided by the general understanding ‘that, at some point, judicial
proceedings must draw to a close and the matter deemed conclusively resolved.’” Northern
States Power Co. v. United States, 79 Fed. Cl. 748, 749 (Fed. Cl. 2007) (quoting Withrow v.
Williams, 507 U.S. 680, 698 (1993)).

       II.     Discussion

        In support of his Motion for Reconsideration and Clarification, petitioner argues just one
of the three bases, i.e., manifest injustice. Petitioner does not argue that there has been a change
in the controlling law or that any previously unavailable evidence has become available.
Petitioner argues that manifest injustice would result if the undersigned did not reconsider her
decision. The undersigned doubts that manifest injustice would result in the dismissal of this
case based on the testimony and evidence in the records. Moreover, in order for a Motion for

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Reconsideration to be granted for manifest injustice, the injustice must be “apparent to the point
of being almost indisputable.” Pac. Gas & Elec. Co., 74 Fed. Cl. at 785. But in the spirit of
liberality, the undersigned clarifies her dismissal decision and thus GRANTS petitioner’s
Motion for Reconsideration and Clarification. However, the reconsideration of the evidence of
testimony in this case still compels the undersigned to DISMISS this case.

        Dr. David Axelrod, petitioner’s expert, is board-certified in allergy and immunology,
rheumatology, and internal medicine. Tr. at 14. He is working part-time seeing patients with
allergy problems. Tr. at 14, 61. About twenty percent of his patients have non-allergy problems.
Tr. at 62.

        Dr. Axelrod testified his “guess” is that the cause of discitis is infection. Tr. at 62-63.
However, he would say discitis was part of an autoimmune disease if someone has ankylosing
spondylitis or granulomatous vasculitis. Tr. at 66. Petitioner does not have ankylosing
spondylitis or granulomatous vasculitis. (Petitioner filed into evidence two case reports dealing
with Wegener’s granulomatosis mimicking a thoracic spondylodiscitis (Exhibit 16) and
spondylodiscitis as the only clinical manifestation of the onset of psoriatic spondyloarthritis
(Exhibit 17). Wegener’s granulomatosus is an autoimmune disease. Just because someone with
Wegener’s can have a condition mimicking spondylodiscitis does not mean that petitioner who
had osteomyelitis/discitis has an autoimmune disease. The second case report dealing with
spondylodiscitis as the only clinical manifestation of the onset of psoriatic spondyloarthritis
interestingly involved treatment of the patient’s spondylodiscitis with antibiotics because the
patient’s doctors regarded the cause as bacterial infection.)

       Dr. Axelrod stated numerous times during his testimony that he is not an infectious
disease expert. The fact that he trained as an immunologist, however, does not mean that all
diseases are immunological. The strong impression the undersigned has is that the only way he
could assist petitioner as his expert was to ignore his guess that the cause of discitis is infection
and assert that petitioner had an autoimmune reaction to his flu vaccination.

        Dr. Axelrod testified, "[Petitioner] had had the flu vaccine. We don't have proof of any
other cause." Tr. at 46. However, it is clear that petitioner’s treating doctors thought the cause
of petitioner’s discitis was an infection. According to Dr. Jeffrey A. Salkin who wrote the
discharge summary before petitioner left Lawrence & Memorial Hospital on October 11, 2013
against medical advice, his treating doctors, said the plan for dealing with petitioner’s discitis
was intravenous antibiotics, an infectious disease consult, and a special procedure biopsy. Med.
recs. Ex. 7, at 15. Petitioner said this was inconvenient for him and walked out of the hospital.

        The Federal Circuit in Capizzano emphasized that the special masters are to evaluate
seriously the opinions of petitioner’s treating doctors since “treating physicians are likely to be in
the best position to determine whether a logical sequence of cause and effect show[s] that the
vaccination was the reason for the injury.” 440 F.3d at 1326. See also Broekelschen v. Sec’y of
HHS, 618 F.3d 1339, 1347 (Fed. Cir. 2010); Andreu v. Sec’y of HHS, 569 F.3d 1367, 1375
(Fed. Cir. 2009). It is thus highly significant to the undersigned that petitioner’s treating doctors

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thought the cause of petitioner’s discitis was an infection and designed a plan to determine which
bacterium was the causing the infection so that they could treat petitioner appropriately with
intravenous antibiotics. Dr. Axelrod’s opinion that the cause of petitioner’s discitis was not an
infection but flu vaccine flies in the face of petitioner’s treating doctors’ opinion. Thus Dr.
Axelrod’s credibility wanes in the face of his assertion.

         Moreover, petitioner’s expert Dr. Axelrod’s opinion that flu vaccine caused petitioner’s
discitis because we do not have proof of any other cause is legally insufficient to make a prima
facie case because his statement does not constitute affirmative evidence of vaccine causation.
See Grant v. Sec’y of HHS, 956 F.2d 1144, 1149 (Fed. Cir. 1992).

        Attempting to offer affirmative proof of causation, Dr. Axelrod testified that there are
hemagglutinin and neuraminidase in flu vaccine that have components similar to cartilage in the
spinal discs, one of the components being GM3. The undersigned asked how much homology of
these components with cartilage is necessary in order to cause someone’s cartilage to be
inflamed. Tr. at 48. Dr. Axelrod said, "I don't have the answer to that. I don't know." Id. His
inability to answer this question also causes Dr. Axelrod’s credibility to wane.

        As for petitioner’s assertion in his Motion for Reconsideration and Clarification that he
was prescribed low dose prednisone and this was a treatment for autoimmune disease, thus
proving he had autoimmune disease, Dr. Axelrod contradicted himself as to whether petitioner
had autoimmune disease. The undersigned said to Dr. Axelrod, "I take it that Mr. Purvis did not
have an autoimmune disease." Tr. at 49. Dr. Axelrod responded: "Not that was indicated in the
chart." Id. However, later on, Dr. Axelrod answered in the affirmative to petitioner’s counsel’s
question that petitioner had "an autoimmune-induced discitis." Tr. at 57. Dr. Axelrod also said
that autoimmune discitis and immune-mediated discitis were the same thing. Tr. at 58, 59-60.
However, none of the treating doctors diagnosed petitioner with autoimmune disease. The
undersigned takes seriously the opinions of petitioner’s treating doctors.

         Dr. Axelrod also said that he was “proposing” the vaccine was the cause of petitioner’s
discitis and “it seemed to involve the L2-L3 disc rather than the other discs,” but he could not
explain why. Tr. at 49-50. This inability further reduces Dr. Axelrod’s credibility.

        Dr. Axelrod admitted that petitioner’s doctors did not do cultures to find out if the cause
of his discitis was bacterial. Tr. at 55.

        Dr. Axelrod said he was unaware of any literature or other support for the conclusion that
an autoimmune or allergic inflammation can cause discitis as a clinically isolated event. Tr. at
70. He also stated that he is unaware of anyone in the medical community who believes that
seasonal flu vaccine causes discitis. Tr. at 86. Dr. Axelrod could not find any literature or cases
reports stating flu vaccine causes discitis. Tr. at 87. Dr. Axelrod stands alone in the medical
community with the theory that flu vaccine causes discitis. This solitary opinion detracts from
Dr. Axelrod’s credibility.


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        Dr. Axelrod agreed with the statement that linear amino acid sequence homology or even
similar conformational structure between an exogenous agent and a self-antigen alone are not
sufficient to prove that molecular mimicry is the pathogenic mechanism for disease. Tr. at 80-
81. This means that Dr. Axelrod’s proffered theory of homology of some undetermined amount
between the hemagglutinin and neuraminidase in flu vaccine and cartilage is not sufficient to
prove that flu vaccine causes disease by homology.

        Dr. Axelrod also agreed with the statement that many such homologies exist and the vast
majority of these are not associated with biologically relevant autoimmune phenomena or actual
human disease. Tr. at 81. This means that Dr. Axelrod’s proffered theory of homology is further
gutted because most likely, assuming any amount of homology, the homology is biologically
irrelevant.

        Dr. Axelrod admitted that petitioner's course of prednisone was ineffective. Tr. at 88.
Petitioner’s assertion in his Motion for Reconsideration and Clarification emphasizes the
importance of petitioner’s doctor prescribing prednisone, but if indeed petitioner had an
autoimmune disease, the prednisone had no effect on this purported autoimmune disease. On the
other hand, petitioner received numerous antibiotics: Ciproflox, Keflex, and Cephalexin, and got
better afterwards. Tr. at 89. Antibiotics are used to treat infections, not autoimmune diseases.
The preferential treatment for petitioner, as both experts agreed, was intravenous antibiotics, but
petitioner refused that treatment.

        Dr. Axelrod testified that he does not think one can prove a causal relationship between
petitioner's influenza vaccine and discitis because there is no reasonable evidence. Tr. at 91. He
stated:

               Well, I don’t think you can prove it. You don’t have – you know,
               you have to have some reasonable evidence, you know, in the data
               that looks at this particular problem in the face of it. So all you can
               really do is look at peripherally things that might connect to each
               other that might cause the problem.

Tr. at 91. In other words, Dr. Axelrod’s opinion is based on speculation, which he admits by use
of the word “might” which means “possible.” Petitioner’s burden is to prove a prima facie case
by preponderant evidence. 42 U.S.C. § 300aa-13(a)(1)(A). “Preponderance” means “more
likely than not” or “probable.” “Preponderance” does not mean “possible.”

       The impression the undersigned got from Dr. Axelrod’s testimony is that he was
markedly uncomfortable, shifting from a theory of central nervous system attack to peripheral
nervous system attack and trying, with a paucity of evidence, to create some theory he could
espouse.




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        Dr. Axelrod defined an autoimmune reaction as a reaction to one's own body. Tr. at 98.
He said that if someone had a vaccine reaction, it is autoimmune because his or her body is
fighting itself. Tr. at 109.

        Dr. Axelrod said part of the problem is that we do not have serial films to know what
really happened in this case and to understand the time course of petitioner's discitis. Tr. at 99.

       Dr. Axelrod recognized that petitioner was on heavy pain medications prior to his
vaccination and that narcotics tend to become less and less beneficial. Tr. at 100.

        Dr. Axelrod's first expert report in this case stated that his opinion was based on the flu
vaccine producing cytokines which breached the blood-brain barrier and entered the brain,
resulting in an increase in vasculature, although it can get into the disc just from the bloodstream
because then you do not have to deal with the blood-brain barrier. Tr. at 102. Dr. Axelrod said
the increased vasculature was likely related to the degenerative spinal disease petitioner had. Id.
He said more blood vessels and increased nerves allowed the cytokines to get to the spine. Tr. at
103.

        Respondent's expert Dr. Collins was a paragon of clarity compared to Dr. Axelrod’s
muddled testimony. She was alert, lucid, and well-qualified to deal with the issues in the case.
She is board-certified in infectious diseases. Tr. at 113. She sees patients, teaches, and does
research. Tr. at 114. She teaches infectious disease to first and second year medical students and
at the wards of a hospital. Tr. at 115.

        Dr. Collins testified that a vertebral disc is quite separated from nerve tissue. Tr. at 119.
She said nerve tissue is only in the spinal cord and leaves the spinal cord out the sides. It goes on
either side of the disc, not through the disc. Id.

        Dr. Collins said that before petitioner received flu vaccine, his disc degeneration was out
of proportion to normal aging. Tr. at 121. The most common type of discitis is an infectious
process in which bacteria in the bloodstream seed the vertebral bodies. Tr. at 122. It is not a
specific attack of a component of the disc, but the presence of bacteria and immune cells fighting
the bacteria that appears as discitis. Id. The vertebral discs do not contain neural tissue from the
spinal cord or from the nerve roots. Tr. at 125.

        Dr. Collins said that Dr. Axelrod's thesis that an autoimmune process adversely affected
petitioner's central nervous system and then affected his disc makes no sense. Tr. at 126. If Dr.
Axelrod proposes that the flu vaccine activated microglia to affect the central nervous system,
then it would not affect the disc because the disc is not part of the central nervous system and
does not have nerves from the central nervous system in it. Id.

        Dr. Collins said petitioner’s spinal MRI analysis was that he had osteomyelitis/discitis.
Tr. at 127. Vertebral osteomyelitis refers to infection of the vertebral bone. Discitis refers to
spread of that infection to the disc. Id. There was abnormal irregularity of the endplates of L2-
L3. Id. Dr. Collins’ opinion is that these MRI findings indicate an osteomyelitis/discitis of an
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infectious origin. Tr. at. 129. That was petitioner’s treating doctors’ opinion as well. Id.

         Dr. Collins said that the approach the doctors took at Lawrence & Memorial Hospital on
October 11, 2013 was “the exact approach one would take for somebody who had
osteomyelitis/discitis.” Tr. at 131. That would be admitting the patient, starting intravenous
antibodies, and biopsying the tissue. Id. They proposed that procedure, given petitioner’s
lumbar spine MRI, because the doctors thought petitioner had an infectious process. Id. Medical
literature supports the conclusion that vertebral discitis and osteomyelitis have an infectious
pathology. Id. Dr. Collins said that usually vertebral osteomyelitis refers to a bacterial infection,
not an autoimmune condition. Tr. at 132.

        As for Exhibit 16 which petitioner submitted showing Wegener’s granulomatosis
mimicking a thoracic spondylodiscitis, Dr. Collins said the lesions in that case were not classic
where one has an adjacent vertebral body endpoint degradation with an effect on the
intervertebral disc that is between those two vertebral bodies. Id. In Exhibit 16, the authors state
the morphology and signal intensity of the vertebral bodies and intervertebral discs were within
normal limits. Tr. at 133. Dr. Collins said that excluded a diagnosis of vertebral osteomyelitis or
spondylodiscitis. In that case report, the authors were not even saying this was a discitis. Id.

        As for Exhibit 17 which petitioner submitted showing spondylodiscitis as the only
clinical manifestation of the onset of psoriatic spondyloarthritis, Dr. Collins said again the MRI
findings were not classic for osteomyelitis/discitis as can be seen in petitioner’s lumbar spinal
MRI. Id. The case report constituting Exhibit 17 depicts a spinal MRI showing partial
hyperintensity, compatible with bone marrow edema, which Dr. Collins said is not the same type
of findings as in the osteomyelitis/discitis that petitioner had. Id. Despite the MRI findings in
the case report constituting Exhibit 17, the patient’s doctors were sure this was probably the
result of an infection because they treated the patient with antibiotics for three months, and the
doctors said that spondylodiscitis usually represents a complication of sepsis and the most
involved pathogens are staph aureus in 60 percent of cases and enterobacter in 30 percent of
cases. Tr. at 134. The authors of the case report constituting Exhibit 17 describe this patient as
having an unusual presentation of discitis. Id. at 134.

         Dr. Collins testified that if petitioner’s discitis were autoimmune, she would expect his
discitis to be part of a greater syndrome rather than a clinically isolated event. Id. In the two
case reports constituting Exhibits 16 and 17, the observation of the spinal cord changes in one
case and the soft tissue changes next to the cord in the other case were both part of larger
syndromes, i.e., Wegener’s granulomatosis (Exhibit 16) and psoriatic spondyloarthritis (Exhibit
17). Id.

        Dr. Collins testified that, in most cases of discitis, it is common to fail to detect the
infection causing it. Tr. at 135. Respondent’s Exhibit A, tab 3, “Pyogenic osteomyelitis of the
spine in the elderly,” notes that in the ten cases that the authors discuss, only one patient had an
unequivocal source of osteomyelitis while two other patients had known recent infectious
illnesses. Tr. at 135, 136. Dr. Collins said that petitioner had recurrent prostatitis which might

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increase his risk of seeding a vertebral body and developing osteomyelitis/discitis. Tr. at 137.
She noted that petitioner has had multiple infectious sources and it is hard to pinpoint a particular
infectious source in this case. Tr. at 140. Petitioner received antibiotic therapy for 32 days,
some of which achieved levels equal to intravenous antibiotics, in particular, Cipro. Tr. at 140,
192. He was also on Keflex for a long time. Tr. at 142. Dr. Collins stated it was impressive
how petitioner always seemed to have some infection or other. Tr. at 144-45.

         Dr. Collins agreed that petitioner’s having multiple infections and re-infections indicates
a vulnerability to having osteomyelitis/discitis. Tr. at 146. She stated the more times bacteria
were introduced into petitioner’s bloodstream, the greater the likelihood he would have a
damaged bone. Id. She also stated that because petitioner’s anatomy is broken down in his spine
due to degenerative disc disease, that “weak link” increases the likelihood of infection. Id.
Although Dr. Collins did not select the particular infection that caused petitioner’s discitis, she
did list the multiple bacterial sources that petitioner had: recurrent prostatitis, skin and soft tissue
infections, chronic sinusitis, respiratory infections, and chronic back pain. Tr. at 147-48.

        Dr. Collins also stated she is not sure when the onset of petitioner’s discitis was. Tr. at
148. In someone with chronic back pain, it is really hard to pinpoint exactly when the discitis
happened. Id. She said she does not know how petitioner’s expert Dr. Axelrod would know that
the onset of petitioner’s osteomyelitis/discitis was August 18, 2013. Id. Dr. Collins said that
petitioner had back pain before August 18, 2013. Id. The onset could have been prior to the
acute worsening. Tr. at 148-49. When the worsening of the back pain began might be hard to
pinpoint in someone, such as petitioner, who always has back pain and who was taking
oxycodone, methadone, and Flexeril all the time. Tr. at 149. These drugs would knock out
petitioner’s ability to recognize the acuteness of his pain. Id. Dr. Collins said that petitioner’s
drugs could have masked the symptoms of his back pain for some time. Tr. at 150. She stated
the onset of petitioner’s discitis absolutely could have preceded his flu vaccination. Id.

        Dr. Collins rejected an autoimmune hypothesis because there is no evidence of neural
involvement in the inflammation of petitioner’s spine, psoas muscle, and vertebral discs. Tr. at
153. Dr. Collins said she is unaware of any similarities between the hemagglutinin and
neuraminidase proteins and GM3 gangliosides. Tr. at 156. Dr. Collins disagreed with Dr.
Axelrod’s theory that cytokines breached the blood-brain barrier to cause local damage to
petitioner’s spine. Tr. at 157. If this had happened, she said petitioner would have had a
systemic response, involving his whole body, and not just one localized site, the disc, because
the disc does not have a blood-brain barrier. Id.

        Dr. Collins testified that she was unaware of any significant homology that would induce
an autoimmune response between neuraminidase and hemagglutinin and neural structures. Tr. at
158-59. She was also unaware of any homology between the two proteins, hemagglutinin and
neuraminidase, in the flu vaccine and tissue in the vertebral disc that would cause an
autoimmune response against the disc. Tr. at 159. Dr. Collins said that someone could do Blast
searches on a computer and find homologies against different proteins all over the place and they
do not cause disease. Id. You can find an immune response against peptides that have

                                                   8
homology to human proteins, but the presence of those antibodies in cells does not correlate with
disease because those people do not have any symptoms. Tr. at 159-60. Those antibodies are
not present in a greater amount in many studies in normal controls compared to people who have
a disease. Tr. at 160.

        Dr. Collins testified that the timing of an autoimmune condition after an inciting
pathogen is introduced into a host body would be a week to 10 days. Tr. at 161. Having
received prior flu vaccines would not have quickened the reaction to one day. Tr. at 163. Dr.
Collins explained that a memory response would be composed of quiescent cells because they
have not been responding actively to the antigens between the time of the earlier vaccination and
the current vaccination. Id. There could be a slightly more rapid increase in cells but not to the
point of a one-day onset. Tr. at 164. Even a three-day onset would be quicker than Dr. Collins
would expect. Tr. at 163-64. Dr. Collins thinks that petitioner’s acute exacerbation of back pain
is purely coincidental to his flu vaccination. Tr. at 167. She thinks it very unlikely that the
vaccine was responsible for petitioner’s condition. Tr. at 167-68. Dr. Collins stated she believes
an infectious process rather than a vaccine reaction is much more likely to have caused
petitioner’s osteomyelitis/discitis. Tr. at 168.

        Petitioner states in his Motion for Reconsideration and Clarification that he is concerned
the undersigned dismissed the case because he failed to show sequelae lasting more than six
months. Mot. at 3 (the pages are not numbered; the undersigned is relying on the pagination that
CM-ECF supplies). The undersigned did not dismiss the petition based on the back pain not
lasting more than six months. Dec. at 11. The undersigned interpreted the medical records to
show that petitioner’s abdominal and testicular pain did not last more than six months. Petitioner
quotes a record from a physical therapist dated April 15, 2014 (more than six months after the
August 15, 2013 flu vaccination) saying that petitioner was there for pain in his central low back.
Mot. at 5. Even though the physical therapist lists the history petitioner gave of having pain in
his back, abdomen, and testicles the day after he received a flu shot in August 2013, it was not
the undersigned’s impression that petitioner was seeking physical therapy for his abdomen and
testicles on April 15, 2014, but that he was seeking physical therapy for his central low back pain
on April 15, 2014.

        In light of the above discussion, the undersigned has reconsidered all the evidence and
clarified the reasons underlying her decision of July 25, 2017. The undersigned considers the
decision dismissing petitioner’s case to be reasonable based on the evidence. Petitioner’s expert
Dr. Axelrod was not as credible as respondent’s expert Dr. Collins. The contemporaneous
medical records support Dr. Collins’ view that a bacterial infection caused petitioner’s
osteomyelitis/discitis and his treating physicians gave him 32 days of antibiotic treatment for it.
They would have given him intravenous antibiotics and done testing and a biopsy if petitioner
had been willing to participate. Because he walked out of the hospital without the recommended
procedures, no one will ever know which bacterium infected his lumbar discs. This hardly
equates to a conclusion that petitioner never had an infectious cause for his discitis, an opinion
petitioner has asserted throughout this case and during the trial, even in Dr. Axelrod’s testimony.


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        Dr. Axelrod’s testimony is also deficient in that there is no credible evidence that
petitioner ever had an autoimmune disease or that flu vaccine played any part in his symptoms
beginning one day after vaccination. Dr. Axelrod agreed that the medical community and the
medical literature do not support his thesis that flu vaccine causes discitis. His assertion that an
autoimmune analysis is pertinent to petitioner’s discitis is the proverbial attempt to put a square
peg into a round hole. Dr. Axelrod started with an analysis which the undersigned suspects he
has used in numerous cases in the Vaccine Program and invented an autoimmune illness where
none exists.

       This case is still DISMISSED.


IT IS SO ORDERED.


Dated: August 18, 2017                                          /s/ Laura D. Millman
                                                                    Laura D. Millman
                                                                     Special Master




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